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File: /home/orthodiedw/nutridatabase/application/controllers/Articles.php
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Effets bénéfiques de l'acide ascorbique dans le traitement de la fibrose pulmonaire induite par le paraquat.

Le paraquat (PQ) est l’un des herbicides les plus largement utilisés dans le monde et provoque des effets toxiques graves chez les humains et les animaux. Une exposition au QP peut conduire à une fibrose pulmonaire (PF) et les mécanismes semblent être liés au stress oxydatif, bien que d'autres voies aient été suggérées. Les antioxydants peuvent être utiles en tant que thérapie, bien que les interventions avec ce type de système restent controversées. Par conséquent, cette étude a examiné le rôle de l’acide ascorbique (vitamine C) après le traitement sur le PF induit par le PQ chez la souris mâle C57 / BL6. La fibrose pulmonaire a été induite par une injection unique de PQ (10 mg / kg; i.p.). Le groupe de contrôle a reçu un véhicule PQ. Sept jours après les injections de PQ ou de véhicule, les souris ont reçu de la vitamine C (150 mg / kg, ip, une fois par jour) ou le véhicule, au cours des 7 jours suivants. Vingt-quatre heures après la dernière dose de vitamine C ou du véhicule, les souris ont été euthanasiées et leur fluide de lavage broncho-alvéolaire (BALF) et leurs poumons ont été recueillis. Les données obtenues ont montré que la vitamine C réduisait le recrutement cellulaire, la sécrétion de cytokine IL-17-a impliquée dans la migration des neutrophiles, le médiateur pro-fibrotique TGF-bêta et le dépôt de collagène. De plus, la vitamine C a élevé les niveaux de superoxyde dismutase (SOD) et de catalase, deux enzymes antioxydantes, mais n'a pas altéré la réponse contractile trachéale évoquée par la méthacholine. Par conséquent, les chercheurs ont souligné les mécanismes non invasifs de la vitamine C et l'ont suggérée comme un outil prometteur pour le traitement de la fibrose pulmonaire induite par une intoxication PQ.


Source: https://www.ncbi.nlm.nih.gov/pubmed/30395570


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